LOW-LEVEL LASER THERAPY FOR WOUND HEALING
LLLT, when used appropriately, can stimulate the healing of injured tissues such as those of the dermis. The mechanisms involved have shown that many of the types of cells whose interaction results in dermal repair can be affected in a therapeutically advantageous manner by treatment with LLLT both in-vitro and in-vivo. Mast cells and macrophages can be stimulated to release growth factors and other substances, whereas the proliferation of fibroblasts, endothelial cells, and keratinocytes maintained in adverse conditions can also be stimulated. The development of granulation tissue is mainly controlled by growth factors released from macrophages. Wound healing involves the following phases:
• Hemostasis: platelets, endothelial cells, fibrin, and fibronectin act through growth factors and cytokines.
• Inflammation: blood clots form, bacteria are attacked, and there is an orderly recruitment of key cells into the wound site.
• Proliferation: cells necessary for wound closure multiply at the wound site to make new tissue and blood vessels.
• Remodeling: the wound is healed and the initial scar tissue is restructured.
Any device that can accelerate any of these processes (transition from hematoma to fibroplasia, development of new blood vessels, production of collagen, or even the remodeling process) could accelerate the healing process of wounds.
Literature indicates that laser photo-bioactivation accelerates inflammation, modulates the level of prostaglandin, enhances the action of macrophages, promotes fibroblast proliferation, facilitates collagen synthesis, fosters immunity, and even accelerates the healing process.
With LLLT we treat
- Gangrene (Dry/Wet)
- Post-surgical wound
- Bedsores
- Fungal infections
- Bruises